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참고문헌 (102)

1. Barker, D. J., "Fetal origins of adult disease: strength of effects and biological basis", International Journal of Epidemiology, 31(6), pp.1235~1239 (2002)
2. Barker, D. J., "In utero programming of chronic disease", Clinical Science, 95(2), pp.115~128 (1998)
3. Tang, W. Y., "Epigenetic reprogramming and imprinting in origins of disease", Reviews in Endocrine & Metabolic Disorders, 8(2), pp.173~182 (2007)
4. Barker, D. J., "Fetal origins of coronary heart disease", British Medical Journal, 311(6998), pp.171~174 (1995)
5. Barker, D. J., "Growth in utero, blood pressure in childhood and adult life, and mortality from cardiovascular disease", British Medical Journal, 298(6673), pp.564~567 (1989)
6. Hales, C. N., "F etal a nd inf ant growth and impaired glucose tolerance at age 64", British Medical Journal, 303(6809), pp.1019~1022 (1991)
7. Bolin, C. M., "Exposure to lead and the developmental origin of oxidative DNA damage in the aging brain", FASEB Journal, 20(6), pp.788~790 (2006)
8. Dolinoy, D. C., "Maternal nutrient supplementation counteracts bisphenol A-induced DNA hypomethylation in early development", Proceedings of the National Academy of Sciences of the United States of America, 104(32), pp.13056~13061 (2007)
9. Krause, E. T., "Early fasting is long lasting: differences in early nutritional conditions reappear under stressful conditions in adult female zebra finches", Public Library of Science ONE, 4(3), pp.e5015~ (2009)
10. Wu, J., "Alzheimer's disease (AD)-like pathology in aged monkeys after infantile exposure to environmental metal lead (Pb): evidence for a developmental origin and environmental link for AD", Journal of Neuroscience, 28(1), pp.3~9 (2008)
11. Gluckman, P. D., "Effect of in utero and early-life conditions on adult health and disease", New England Journal of Medicine, 359(1), pp.61~73 (2008)
12. Waggoner, D, "Mechanisms of disease: epigenesis", Seminars in Pediatric Neurology, 14(1), pp.7~14 (2007)
13. Waterland, R. A, "Epigenetic epidemiology of the developmental origins hypothesis", Annual Review of Nutrition, 27, pp.363~388 (2007)
14. Esteller, M, "Epigenetics in cancer", New England Journal of Medicine, 358(11), pp.1148~1159 (2008)
15. Lopez, J, "The context and potential of epigenetics in oncology", British Journal of Cancer, 100(4), pp.571~577 (2009)
16. Yoo, C. B, "Epigenetic therapy of cancer: past, present and future", Nature Reviews Drug Discovery, 5(1), pp.37~50 (2006)
17. Simmons, R. A, "Developmental origins of beta-cell failure in type 2 diabetes: the role of epigenetic mechanisms", Pediatric Research, 61(5), pp.64R~67R (2007)
18. Campion, J, "Individuality and epigenetics in obesity", Obes Rev, 10(4), pp.383~392 (2009)
19. Zawia, N. H, "Epigenetics, oxidative stress, and Alzheimer disease", Free Radical Biology & Medicine, 46(9), pp.1241~1249 (2009)
20. Schanen, N. C, "Epigenetics of autism spectrum disorders", Human Molecular Genetics, 15(2), pp.R138~R150 (2006)
21. Steinke, J. W, "5. Genetics of allergic disease", Journal of Allergy and Clinical Immunology, 121(2), pp.S384~S387 (2008)
22. Foley, D. L, "P rospects f or epigenetic epidemiology", American Journal of Epidemiology, 169(4), pp.389~400 (2009)
23. Holliday, R, "Epigenetics: a historical overview", Epigenetics, 1(2), pp.76~80 (2006)
24. Allis, C. D, "Overview and Concepts. in: Epigenetics" (2007), Cold Spring Harbor Laboratory Press
25. Robertson, K. D, "DNA methylation in health and disease", Nature Reviews Genetics, 1(1), pp.11~19 (2000)
26. Slotkin, R. K, "Transposable elements and the epigenetic regulation of the genome", Nature Reviews Genetics, 8(4), pp.272~285 (2007)
27. Zeisel, S. H, "Importance of methyl donors during reproduction", American Journal of Clinical Nutrition, 89(2), pp.673S~677S (2009)
28. Vickers, M. H, "Neonatal leptin treatment reverses developmental programming", Endocrinology, 146(10), pp.4211~4216 (2005)
29. Waterland, R. A, "Transposable elements: targets for early nutritional effects on epigenetic gene regulation", Molecular and Cellular Biology, 23(15), pp.5293~5300 (2003)
30. Santos, F, "Dynamic reprogramming of DNA methylation in the early mouse embryo", Developmental Biology, 241(1), pp.172~182 (2002)
31. Kaneko-Ishino, T, "hypothesis: the necessity of a monoallelic gene expression mechanism in mammalian development", Cytogenetic and Genome Research, 113(1), pp.24~30 (2006)
32. Bird, A, "Perceptions of epigenetics", Nature, 447(7143), pp.396~398 (2007)
33. Kaati, G, "Cardiovascular and diabetes mortality determined by nutrition during parents' and grandparents' slow growth period", European Journal of Human Genetics, 10(11), pp.682~688 (2002)
34. Cheng, R. Y, "Epigenetic and gene expression changes related to transgenerational carcinogenesis", Molecular Carcinogenesis, 40(1), pp.1~11 (2004)
35. Anway, M. D, "Endocrine disruptor vinclozolin induced epigenetic transgenerational adult-onset disease", Endocrinology, 147(12), pp.5515~5523 (2006)
36. Ercal, N, "Toxic metals and oxidative stress part I: mechanisms involved in metal-induced oxidative damage", Current Topics in Medicinal Chemistry, 1(6), pp.529~539 (2001)
37. Bindhumol, V, "Bisphenol A induces reactive oxygen species gener ation in the liver of male rats", Toxicology, 188(2), pp.117~124 (2003)
38. Brook, R. D, "Air pollution and cardiovascular disease: a statement for healthcare professionals from the Expert Panel on Population and Prevention Science of the American Heart Association", Circulation, 109(21), pp.2655~2671 (2004)
39. Franco, R, "Oxidative stress, DNA methylation and carcinogenesis", Cancer Letters, 266(1), pp.6~11 (2008)
40. Xu, Y, "Association of oxidative stress with arsenic methylation in chronic arsenic-exposed children and adults", Toxicology and Applied Pharmacology, 232(1), pp.142~149 (2008)
41. Prins, G. S, "Ho and S. M. Perinatal exposure to oestradiol and bisphenol A alters the prostate epigenome and increases susceptibility to carcinogenesis", Basic Clin Pharmacol Toxicol, 102(2), pp.134~138 (2008)
42. Valinluck, V, "Oxidative damage to methyl-CpG sequences inhibits the binding of the methyl-CpG binding domain (MBD) of methyl-CpG binding protein 2 (MeCP2)", Nucleic Acids Research, 32(14), pp.4100~4108 (2004)
43. Poirier, L. A, "The prospective role of abnormal methyl metabolism in cadmium toxicity", Environmental Health Perspectives, 10(5), pp.793~795 (2002)
44. Reichard, J. F, "Long term low-dose arsenic exposure induces loss of DNA methylation", Biochemical and Biophysical Research Communications, 352(1), pp.188~192 (2007)
45. Takiguchi, M., "Effects of cadmium on DNA- (Cytosine-5) methyltransferase activity and DNA methylation status during cadmium-induced cellular transformation", Experimental Cell Research, 286(2), pp.355~365 (2003)
46. Chanda, S, "DNA hypermethylation of promoter of gene p53 and p16 in arsenic-exposed people with and without malignancy", Toxicological Science, 89(2), pp.431~437 (2006)
47. Goering, P. L, "The enigma of arsenic carcinogenesis: role of metabolism", Toxicological Science, 49(1), pp.5~14 (1999)
48. Sutherland, J. E, "Epigenetics and the environment", Annals of the New York Academy of Sciences, 983, pp.151~160 (2003)
49. ATSDR, "Toxicological profile for cadmium. Atlanta" (1999), Department of Health & Human Services
50. Waalkes, M. P, "Cadmium carcinogenesis in review", Journal of Inorganic Biochemistry, 79(1), pp.241~244 (2000)
51. Huang, D, "Global DNA hypomethylation, rather than reactive oxygen species (ROS), a potential facilitator of cadmium- stimulated K562 cell proliferation", Toxicology Letters, 179(1), pp.43~47 (2008)
52. Jiang, G, "Effects of long-term low-dose cadmium exposure on genomic DNA methylation in human embryo lung fibroblast cells", Toxicology, 244(1), pp.49~55 (2008)
53. Benbrahim-Tallaa, L, "Tumor suppressor gene inactivation during cadmium-induced malignant transformation of human prostate cells correlates with overexpression of de novo DNA methyltransferase", Environmental Health Perspectives, 115(10), pp.1454~1459 (2007)
54. Benbrahim-Tallaa, L, "Molecular events associated with arsenicinduced malignant transformation of human prostatic epithelial cells: aberrant genomic DNA methylation and K-ras oncogene activation", Toxicology and Applied Pharmacology, 206(3), pp.288~298 (2005)
55. Chen, H, "Association of cmyc overexpression and hyperproliferation with arsenite- induced malignant transformation", Toxicology and Applied Pharmacology, 175(3), pp.260~268 (2001)
56. Sciandrello, G., "Arsenic-induced DNA hypomethylation affects chromosomal instability in mammalian cells", Carcinogenesis, 25(3), pp.413~417 (2004)
57. Chai, C. Y., "Arsenic salts induced autophagic cell death and hypermethylation of DAPK promoter in SV-40 immortalized human uroepithelial cells", Toxicology Letters, 173(1), pp.48~56 (2007)
58. Chen, W. T., "Urothelial carcinomas arising in arsenic-contaminated areas are associated with hypermethylation of the gene promoter of the deathassociated protein kinase", Histopathology, 51(6), pp.785~792 (2007)
59. Cui, X., "Chronic oral exposure to inorganic arsenate interferes with methylation status of p16INK4a and RASSF1A and induces lung cancer in A", J mice, 2006, pp.91(2), 372~381
60. Zhang, A. H., "Analysis of p16 gene mutation, deletion and methylation in patients with arseniasis produced by indoor unventilated-stove coal usage in Guizhou, China", Journal of Toxicology and Environmental Health, 70(11), pp.970~975 (2007)
61. Ramirez, T, "Sodium arsenite modulates histone acetylation, histone deacetylase activity and HMGN protein dynamics in human cells", Chromosoma, 117(2), pp.147~157 (2008)
62. Pilsner, J. R, "Genomic methylation of peripheral blood leukocyte DNA: influences of arsenic and folate in Bangladeshi adults", American Journal of Clinical Nutrition, 86(4), pp.1179~1186 (2007)
63. Pilsner, J. R, "Folate deficiency, hyperhomocysteinemia, low urinary creatinine, and hypomethylation of leukocyte DNA are risk factors for arsenic-induced skin lesions", Environmental Health Perspectives, 117(2), pp.254~260 (2009)
64. Kanduc, D, "Transitory DNA hypomethylation during liver cell proliferation induced by a single dose of lead nitrate", Archives of Biochemistry and Biophysics, 286(1), pp.212~216 (1991)
65. Rossiello, M. R, "DNA hypomethylation during liver cell proliferation induced by a single dose of lead nitrate", Bollettino Societa Italiana Biologia Sperimentale, 67(12), pp.993~997 (1991)
66. Basha, M. R, "The fetal basis of amyloidogenesis: exposure to lead and latent overexpression of amyloid precursor protein and beta-amyloid in the aging brain", Journal of Neuroscience, 25(4), pp.823~829 (2005)
67. Pilsner, J. R, "Inf luence o f Prenatal Lead Exposure on Genomic Methylation of Cord Blood DNA", Environmental Health Perspectives (2009)
68. Ji, W, "Epigenetic silencing of O6-methylguanine DNA methyltransferase gene in NiS-transformed cells", Carcinogenesis, 29(6), pp.1267~1275 (2008)
69. Ke, Q, "Alterations of histone modifications and transgene silencing by nickel chloride", Carcinogenesis, 27(7), pp.1481~1488 (2006)
70. Lee, Y. W, "Carcinogenic nickel silences gene expression by chromatin condensation and DNA methylation: a new model for epigenetic carcinogens", Molecular and Cellular Biology, 15(5), pp.2547~2557 (1995)
71. Kondo, K, "The reduced expression and aberrant methylation of p16(INK4a) in chromate workers with lung cancer", Lung Cancer, 53(3), pp.295~302 (2006)
72. Schnekenburger, M, "Chromium cross-links histone deacetylase 1-DNA methyltransferase 1 complexes to chromatin, inhibiting histone-remodeling marks critical for transcriptional activation", Molecular and Cellular Biology, 27(20), pp.7089~7101 (2007)
73. "NTP, CERHR. NTP-CERHR Monograph on the potential human reproductive and developmental effects of bisphenol A. National Institutes of Health 2008", ~ (2008)
74. Maffini, M. V, "Endocrine disruptors and reproductive health: the case of bisphenol-A", Molecular and Cellular Endocrinology, pp.254~255 (2006)
75. Welshons, W. V, "Large effects from small exposures. III. Endocrine mechanisms mediating effects of bisphenol A at levels of human exposure", Endocrinology, 147(6), pp.S56~S69 (2006)
76. Ho, S. M, "Developmental exposure to estradiol and bisphenol A increases susceptibility to prostate carcinogenesis and epigenetically regulates phosphodiesterase type 4 variant 4", Cancer Research, 66(11), pp.5624~5632 (2006)
77. Rubin, M. M, "Antenatal exposure to DES: lessons learned...future concerns", Obstetrical & Gynecological Survey, 62(8), pp.548~555 (2007)
78. Li, S, "Neonatal diethylstilbestrol exposure induces persistent elevation of c-fos expression and hypomethylation in its exon-4 in mouse uterus", Molecular Carcinogenesis, 38(2), pp.78~84 (2003)
79. Li, S, "Developmental exposure to diethylstilbestrol elicits demethylation of estrogen-responsive lactoferrin gene in mouse uterus", Cancer Research, 57(19), pp.4356~4359 (1997)
80. Sato, K, "Neonatal exposure to diethylstilbestrol alters t he e xpression o f DNA m ethyltransferases and methylation of genomic DNA in the epididymis of mice", Endocrine Journal, 53(3), pp.331~337 (2006)
81. Tang, W. Y, "Persistent hypomethylation in the promoter of nucleosomal binding protein 1 (Nsbp1) correlates with overexpression of Nsbp1 in mouse uteri neonatally exposed to diethylstilbestrol or genistein", Endocrinology, 149(12), pp.5922~5931 (2008)
82. Newbold, R. R, "Adverse effects of the model environmental estrogen diethylstilbestrol are transmitted to subsequent generations", Endocrinology, 147(6), pp.S11~S17 (2006)
83. Anway, M. D, "Epigenetic transgenerational actions of endocrine disruptors and male fertility", Science, 308(5727), pp.1466~1469 (2005)
84. Crews, D, "Transgenerational epigenetic imprints on mate preference", Proceedings of the National Academy of Sciences of the United States of America, 104(14), pp.5942~5946 (2007)
85. Rusiecki, J. A, "Global DNA hypomethylation is associated with high serum-persistent organic pollutants in Greenlandic Inuit", Environmental Health Perspectives, 116(11), pp.1547~1552 (2008)
86. Bunger, M. K, "Abnormal liver development and resistance to 2,3,7,8-tetrachlorodibenzo- p-dioxin toxicity in mice carrying a mutation in the DNA-binding domain of the aryl hydrocarbon receptor", Toxicological Science, 106(1), pp.83~92 (2008)
87. Okey, A. B, "An aryl hydrocarbon receptor odyssey to the shores of toxicology: the Deichmann Lecture, International Congress of Toxicology-XI", Toxicological Science, 98(1), pp.5~38 (2007)
88. Wu, Q, "Exposure of mouse preimplantation embryos to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) alters the methylation status of imprinted genes H19 and Igf2", Biology of Reproduction, 70(6), pp.1790~1797 (2004)
89. Schwartz, J, "Air pollution and children's health", Pediatrics, 113(4), pp.1037~1043 (2004)
90. Baccarelli, A, "Rapid DNA methylation changes after exposure to traffic particles", American Journal of Respiratory and Critical Care Medicine, 179(7), pp.572~578 (2009)
91. Perera, F, "R elation of DNA methylation of 5'-CpG island of ACSL3 to transplacental exposure to airborne polycyclic aromatic hydrocarbons and childhood asthma", Public Library of Science ONE, 4(2), pp.e4488~ (2009)
92. Tarantini, L, "Effects o f particulate matter on genomic DNA methylation content and iNOS promoter methylation", Environmental Health Perspectives, 117(2), pp.217~222 (2009)
93. Belinsky, S. A, "Aberrant CpG island methylation of the p16(INK4a) and estrogen receptor genes in rat lung tumors induced by particulate carcinogens", Carcinogenesis, 23(2), pp.335~339 (2002)
94. Park, S. K, "Traffic-related particles are associated with elevated homocysteine: the VA normative aging study", American Journal of Respiratory and Critical Care Medicine, 178(3), pp.283~289 (2008)
95. Cobiac, L, "Epigenomics and nutrition", Forum of Nutrition, 60, pp.31~41 (2007)
96. Gallou-Kabani, C, "Nutri-epigenomics: lifelong remodelling of our epigenomes by nutritional and metabolic factors and beyond", Clinical Chemistry and Laboratory Medicine, 45(3), pp.321~327 (2007)
97. Baccarelli, A, "Cardiac autonomic dysfunction: effects from particulate air pollution and protection by dietary methyl nutrients and metabolic polymorphisms", Circulation, 117(14), pp.1801~1809 (2008)
98. Fish, E. W, "Epigenetic programming of stress responses through variations in maternal care", Annals of the New York Academy of Sciences, 1036, pp.167~180 (2004)
99. Szyf, M, "Maternal care, the epigenome and phenotypic differences in behavior", Reproductive Toxicology, 24(1), pp.9~19 (2007)
100. Weaver, I. C, "Epigenetic programming by maternal behavior", Nature Neuroscience, 7(8), pp.847~854 (2004)
101. Weaver, I. C, "Reversal of maternal programming of stress responses in adult offspring through methyl supplementation: altering epigenetic marking later in life", Journal of Neuroscience, 25(47), pp.11045~11054 (2005)
102. "WHO", FAO. Diet, nutrition and the prevention of chronic diseases. World Health Organ Tech Rep Ser, 916, i-viii, 1-149, backcover, 2003

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