Store-operated Ca2+ entry in muscle physiology and diseases

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참고문헌 (92)

1. Putney, J. W. Jr, "A model for receptor-regulated calcium entry", Cell Calcium, 7, pp.1~12 (1986)
2. Zhang, S. L., "STIM1 is a Ca2+ sensor that activates CRAC channels and migrates from the Ca2+ store to the plasma membrane", Nature, 437, pp.902~905 (2005)
3. Liou, J., "STIM is a Ca2+ sensor essential for Ca2+-store-depletion-triggered Ca2+ influx", Curr. Biol, 15, pp.1235~1241 (2005)
4. Roos, J., "STIM1, an essential and conserved component of store-operated Ca2+ channel function", J. Cell Biol, 169, pp.435~445 (2005)
5. Yeromin, A. V., "Molecular identification of the CRAC channel by altered ion selectivity in a mutant of Orai", Nature, 443, pp.226~229 (2006)
6. Prakriya, M., "Orai1 is an essential pore subunit of the CRAC channel", Nature, 443, pp.230~233 (2006)
7. Vig, M., "CRACM1 is a plasma membrane protein essential for store-operated Ca2+ entry", Science, 312, pp.1220~1223 (2006)
8. Feske, S., "A mutation in Orai1 causes immune deficiency by abrogating CRAC channel function", Nature, 441, pp.179~185 (2006)
9. Luik, R. M., "The elementary unit of store-operated Ca2+ entry: local activation of CRAC channels by STIM1 at ER-plasma membrane junctions", J. Cell Biol, 174, pp.815~825 (2006)
10. Huang, G. N., "STIM1 carboxyl-terminus activates native SOC, I (crac) and TRPC1 channels", Nat. Cell Biol, 8, pp.1003~1010 (2006)
11. Rios, E., "Charge movement and the nature of signal transduction in skeletal muscle excitation-contraction coupling", Annu. Rev. Physiol, 54, pp.109~133 (1992)
12. Kurebayashi, N., "Depletion of Ca2+ in the sarcoplasmic reticulum stimulates Ca2+ entry into mouse skeletal muscle fibres", J. Physiol, 533, pp.185~199 (2001)
13. Pan, Z., "Dysfunction of store-operated calcium channel in muscle cells lacking mg29", Nat. Cell Biol, 4, pp.379~383 (2002)
14. Stiber, J., "STIM1 signalling controls store-operated calcium entry required for development and contractile function in skeletal muscle", Nat. Cell Biol, 10, pp.688~697 (2008)
15. McCarl, C. A., "ORAI1 deficiency and lack of store-operated Ca2+ entry cause immunodeficiency, myopathy, and ectodermal dysplasia", J. Allergy Clin. Immunol, 124, pp.1311~1318 (2009)
16. Picard, C., "STIM1 mutation associated with a syndrome of immunodeficiency and autoimmunity", N. Engl. J. Med, 360, pp.1971~1980 (2009)
17. Lewis, R. S., "Store-operated calcium channels: new perspectives on mechanism and function", Cold Spring Harb. Perspect. Biol, 3, pp.a003970~ (2011)
18. Putney, J. W, "The physiological function of storeoperated calcium entry", Neurochem. Res, 36, pp.1157~1165 (2011)
19. Feske, S, "ORAI1 and STIM1 deficiency in human and mice: roles of store-operated Ca2+ entry in the immune system and beyond", Immunol. Rev, 231, pp.189~209 (2009)
20. Feske, S, "CRAC channelopathies", Pflugers. Arch, 460, pp.417~435 (2010)
21. Feske, S, "Immunodeficiency due to defects in store-operated calcium entry", Ann. N. Y. Acad. Sci, 1238, pp.74~90 (2011)
22. Ma, J., "Retrograde activation of storeoperated calcium channel", Cell Calcium, 33, pp.375~384 (2003)
23. Rios, E., "The mechanical hypothesis of excitation-contraction (EC) coupling in skeletal muscle", J. Muscle. Res. Cell Motil, 12, pp.127~135 (1991)
24. Cota, G., "Voltage-dependent inactivation of slow calcium channels in intact twitch muscle fibers of the frog", J. Gen. Physiol, 94, pp.937~951 (1989)
25. Nakai, J., "Enhanced dihydropyridine receptor channel activity in the presence of ryanodine receptor", Nature, 380, pp.72~75 (1996)
26. MacLennan, D. H, "Ca2+ signalling and muscle disease", Eur. J. Biochem, 267, pp.5291~5297 (2000)
27. Hovnanian, A, "SERCA pumps and human diseases", Subcell. Biochem, 45, pp.337~363 (2007)
28. MacLennan, D. H., "Structure-function relationships in the Ca (2+)-binding and translocation domain of SERCA1: physiological correlates in Brody disease", Acta. Physiol. Scand, 643, pp.55~67 (1998)
29. Periasamy, M., "SERCA pump isoforms: their role in calcium transport and disease", Muscle. Nerve, 35, pp.430~442 (2007)
30. Bannister, R. A., "The skeletal L-type Ca (2+) current is a major contributor to excitation-coupled Ca (2+) entry", J. Gen. Physiol, 133, pp.79~91 (2009)
31. Cherednichenko, G., "Conformational activation of Ca2+ entry by depolarization of skeletal myotubes", Proc. Natl. Acad. Sci. U. S. A, 101, pp.15793~15798 (2004)
32. Zhao, X., "Azumolene inhibits a component of store-operated calcium entry coupled to the skeletal muscle ryanodine receptor", J. Biol. Chem, 281, pp.33477~33486 (2006)
33. Stiber, J. A., "The role of store-operated calcium influx in skeletal muscle signaling", Cell Calcium, 49, pp.341~349 (2011)
34. Brotto, M., "Aging, sarcopenia and store-operated calcium entry: a common link?", Cell Cycle, 10, pp.4201~4202 (2011)
35. Dirksen, R. T, "Checking your SOCCs and feet: the molecular mechanisms of Ca2+ entry in skeletal muscle", J. Physiol, 587, pp.3139~3147 (2009)
36. Yarotskyy, V., "Temperature and RyR1 regulate the activation rate of store-operated Ca (2)+ entry current in myotubes", Biophys J, 103, pp.202~211 (2012)
37. Pan, Z., "Fluorescencebased measurement of store-operated calcium entry in live cells: from cultured cancer cell to skeletal muscle fiber", J. Vis. Exp, 60 (2012)[DOI]
38. Hirata, Y., "Uncoupling store-operated Ca2+ entry and altered Ca2+ release from sarcoplasmic reticulum through silencing of junctophilin genes", Biophys. J, 90, pp.4418~4427 (2006)
39. Launikonis, B. S., "Identification of the coupling between skeletal muscle store-operated Ca2+ entry and the inositol trisphosphate receptor", Proc. Natl. Acad. Sci. U S. A, 100, pp.2941~2944 (2003)
40. Zhao, X., "Orai1 mediates exacerbated Ca (2+) entry in dystrophic skeletal muscle", PLoS One, 7, pp.e49862~ (2012)
41. Launikonis, B. S., "Store-operated Ca2+ entry during intracellular Ca2+ release in mammalian skeletal muscle", J. Physiol, 583, pp.81~97 (2007)
42. Hou, X., "Crystal structure of the calcium release-activated calcium channel Orai", Science, 338, pp.1308~1313 (2012)
43. Louis, M., "TRPC1 regulates skeletal myoblast migration and differentiation", J. Cell Sci, 121, pp.3951~3959 (2008)
44. Brinkmeier, H., "TRP channels in skeletal muscle: gene expression, function and implications for disease", Adv. Exp. Med. Biol, 704, pp.749~758 (2011)
45. Zhao, X., "Compromised store-operated Ca2+ entry in aged skeletal muscle", Aging Cell, 7, pp.561~568 (2008)
46. Lyfenko, A. D, "Differential dependence of store-operated and excitation-coupled Ca2+ entry in skeletal muscle on STIM1 and Orai1", J. Physiol, 586, pp.4815~4824 (2008)
47. Wei-Lapierre, L., "Orai1-dependent calcium entry promotes skeletal muscle growth and limits fatigue", Nat. Commun, 4, pp.2805~ (2013)
48. Ma, J., "Junctional membrane structure and store operated calcium entry in muscle cells", Front. Biosci, 8, pp.242~255 (2003)
49. Flucher, B. E., "Development of the excitation-contraction coupling apparatus in skeletal muscle: association of sarcoplasmic reticulum and transverse tubules with myofibrils", Dev. Biol, 160, pp.135~147 (1993)
50. Darbellay, B., "STIM1L is a new actin-binding splice variant involved in fast repetitive Ca2+ release", J. Cell Biol, 194, pp.335~346 (2011)
51. Mo, C., "Prostaglandin E2: from clinical applications to its potential role in bone- muscle crosstalk and myogenic differentiation", Recent. Pat. Biotechnol, 6, pp.223~229 (2012)
52. Jahn, K., "Skeletal muscle secreted factors prevent glucocorticoid-induced osteocyte apoptosis through activation of beta-catenin", Eur. Cell Mater, 24, pp.197~209 (2012)
53. Takeshima, H., "Mitsugumin29, a novel synaptophysin family member from the triad junction in skeletal muscle", Biochem. J, 331(1), pp.317~322 (1998)
54. Zhao, X., "Molecular architecture of Ca2+ signaling control in muscle and heart cells", Channels (Austin), 5, pp.391~396 (2011)
55. Komazaki, S., "Abnormal formation of sarcoplasmic reticulum networks and triads during early development of skeletal muscle cells in mitsugumin29-deficient mice", Dev. Growth Differ, 43, pp.717~723 (2001)
56. Nishi, M., "Abnormal features in skeletal muscle from mice lacking mitsugumin29", J. Cell Biol, 147, pp.1473~1480 (1999)
57. Takeshima, H., "Junctophilins: a novel family of junctional membrane complex proteins", Mol. Cell, 6, pp.11~22 (2000)
58. Weisleder, N., "Immunoproteomic approach to excitation--contraction coupling in skeletal and cardiac muscle: molecular insights revealed by the mitsugumins", Cell Calcium, 43, pp.1~8 (2008)
59. Nishi, M., "Characterization of human junctophilin subtype genes", Biochem. Biophys. Res. Commun, 273, pp.920~927 (2000)
60. Nishi, M., "Coexpression of junctophilin type 3 and type 4 in brain", Brain Res. Mol. Brain Res, 118, pp.102~110 (2003)
61. Ito, K., "Deficiency of triad junction and contraction in mutant skeletal muscle lacking junctophilin type 1", J. Cell Biol, 154, pp.1059~1067 (2001)
62. Komazaki, S., "Abnormal junctional membrane structures in cardiac myocytes expressing ectopic junctophilin type 1", FEBS Lett, 542, pp.69~73 (2003)
63. Pan, Z., "Co-expression of MG29 and ryanodine receptor leads to apoptotic cell death: effect mediated by intracellular Ca2+ release", J. Biol. Chem, 279, pp.19387~19390 (2004)
64. Jin Seok Woo, "TRPC3 cation channel plays an important role in proliferation and differentiation of skeletal muscle myoblasts", Experimental and Molecular Medicine, 42(9), pp.614~627 (2010), 생화학분자생물학회
65. Woo, J. S., "S165F mutation of junctophilin 2 affects Ca2+ signalling in skeletal muscle", Biochem. J, 427, pp.125~134 (2010)
66. Woo, J. S., "Glutamate at position 227 of junctophilin- 2 is involved in binding to TRPC3", Mol. Cell Biochem, 328, pp.25~32 (2009)
67. Shin, D. W., "A retrograde signal from calsequestrin for the regulation of store-operated Ca2+ entry in skeletal muscle", J. Biol. Chem, 278, pp.3286~3292 (2003)
68. Zhao, X., "Increased store-operated Ca2+ entry in skeletal muscle with reduced calsequestrin- 1 expression", Biophys. J, 99, pp.1556~1564 (2010)
69. Yarotskyy, V., "Accelerated activation of SOCE current in myotubes from two mouse models of anesthetic- and heat-induced sudden death", PLoS One, 8, pp.e77633~ (2013)
70. Allen, D. G., "Skeletal muscle fatigue: cellular mechanisms", Physiol. Rev, 88, pp.287~332 (2008)
71. Zhao, X., "Enhanced resistance to fatigue and altered calcium handling properties of sarcalumenin knockout mice", Physiol. Genomics, 23, pp.72~78 (2005)
72. Kiviluoto, S., "STIM1 as a key regulator for Ca2+ homeostasis in skeletal-muscle development and function", Skelet Muscle, 1, pp.16~ (2011)
73. Darbellay, B., "STIM1- and Orai1-dependent store-operated calcium entry regulates human myoblast differentiation", J. Biol. Chem, 284, pp.5370~5380 (2009)
74. Li, T., "STIM1-Ca (2+) signaling is required for the hypertrophic growth of skeletal muscle in mice", Mol. Cell Biol, 32, pp.3009~3017 (2012)
75. Thornton, A. M., "Store-operated Ca (2+) entry (SOCE) contributes to normal skeletal muscle contractility in young but not in aged skeletal muscle", Aging (Albany NY), 3, pp.621~634 (2011)
76. Zahn, J. M., "Transcriptional profiling of aging in human muscle reveals a common aging signature", PLoS Genet, 2, pp.e115~ (2006)
77. Vanterpool, C. K., "Advancing age alters rapid and spontaneous refilling of caffeine-sensitive calcium stores in sympathetic superior cervical ganglion cells", J. Appl. Physiol, 99, pp.963~971 (2005)
78. Bohm, J., "Constitutive activation of the calcium sensor STIM1 causes tubular-aggregate myopathy", Am. J. Hum. Genet, 92, pp.271~278 (2013)
79. Parness, J., "The myotonias and susceptibility to malignant hyperthermia", Anesth. Analg, 109, pp.1054~1064 (2009)
80. Duke, A. M., "Store-operated Ca2+ entry in malignant hyperthermia-susceptible human skeletal muscle", J. Biol. Chem, 285, pp.25645~25653 (2010)
81. Romanick, M., "Murine models of atrophy, cachexia and sarcopenia in skeletal muscle", Biochim. Biophys. Acta, 1832, pp.1410~1420 (2013)
82. Lowe, D. A., "Myofibrillar myosin ATPase activity in hindlimb muscles from young and aged rats", Mech. Ageing. Dev, 125, pp.619~627 (2004)
83. Lowe, D. A., "Force generation, but not myosin ATPase activity, declines with age in rat muscle fibers", Am. J. Physiol. Cell Physiol, 283, pp.C187~C192 (2002)
84. Romero-Suarez, S., "Muscle-specific inositide phosphatase (MIP", MTMR14) is reduced with age and its loss accelerates skeletal muscle aging process by altering calcium homeostasis, 2010, pp.2, 504~513
85. Russ, D. W., "Evolving concepts on the age-related changes in "muscle quality"", J. Cachexia. Sarcopenia. Muscle, 3, pp.95~109 (2012)
86. Manini, T. M., "Dynapenia and aging: an update", J. Gerontol. A Biol. Sci. Med. Sci, 67, pp.28~40 (2012)
87. Collins, H. E., "STIM1", Orai1-mediated SOCE: current perspectives and potential roles in cardiac function and pathology, 2013, pp.305, H446~H458
88. Touchberry, C. D., "Store-operated calcium entry is present in HL-1 cardiomyocytes and contributes to resting calcium", Biochem. Biophys. Res. Commun, 416, pp.45~50 (2011)
89. Volkers, M., "Orai1 deficiency leads to heart failure and skeletal myopathy in zebrafish", J. Cell Sci, 125, pp.287~294 (2012)
90. Luo, X., "STIM1-dependent store-operated Ca (2) (+) entry is required for pathological cardiac hypertrophy", J. Mol. Cell Cardiol, 52, pp.136~147 (2012)
91. Ohba, T., "Essential role of STIM1 in the development of cardiomyocyte hypertrophy", Biochem. Biophys. Res. Commun, 389, pp.172~176 (2009)
92. Voelkers, M., "Orai1 and Stim1 regulate normal and hypertrophic growth in cardiomyocytes", J. Mol. Cell Cardiol, 48, pp.1329~1334 (2010)

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