Two Types of Mouse Models for Sarcopenia Research: Senescence Acceleration and Genetic Modification Models

키워드

참고문헌 (86)

1. Ian Janssen, "Evolution of sarcopenia research", Applied Physiology, Nutrition, and Metabolism, 35(5), pp.707~712 (2010), Canadian Science Publishing[DOI]
2. "AIM coalition announces establishment of ICD-10-CM code for sarcopenia by the Centers for Disease Control and Prevention" (2016)[URL]
3. Kyung-Wan Baek, "Rodent Model of Muscular Atrophy for Sarcopenia Study", 대한골대사학회지, 27(2), pp.97~110 (2020), 대한골대사학회
4. Wim Derave, "Soleus muscles of SAMP8 mice provide an accelerated model of skeletal muscle senescence", Experimental Gerontology, 40(7), pp.562~572 (2005), Elsevier BV[DOI]
5. T TAKEDA, "A new murine model of accelerated senescence", Mechanisms of Ageing and Development, 17(2), pp.183~194 (1981), Elsevier BV[DOI]
6. Toshio Takeda, "A novel murine model of aging, Senescence-Accelerated Mouse (SAM)", Archives of Gerontology and Geriatrics, 19(2), pp.185~192 (1994), Elsevier BV[DOI]
7. T Takeda, "Pathobiology of the senescence-accelerated mouse (SAM)", Experimental Gerontology, 32(1-2), pp.117~127 (1997), Elsevier BV[DOI]
8. Chiyoko Hirofuji, "SDH activity and cell size of tibialis anterior motoneurons and muscle fibers in SAMP6", NeuroReport, 11(4), pp.823~828 (2000), Ovid Technologies (Wolters Kluwer Health)[DOI]
9. Tomofumi Nishikawa, "Tubular aggregates in the skeletal muscle of the senescence-accelerated mouse; SAM", Mechanisms of Ageing and Development, 114(2), pp.89~99 (2000), Elsevier BV[DOI]
10. Toshio Takeda, "Senescence-accelerated mouse (SAM): a biogerontological resource in aging research", Neurobiology of Aging, 20(2), pp.105~110 (1999), Elsevier BV[DOI]
11. Yoichi Chiba, "The Senescence-accelerated Mouse (SAM): A Higher Oxidative Stress and Age-dependent Degenerative Diseases Model", Neurochemical Research, 34(4), pp.679~687 (2009), Springer Science and Business Media LLC[DOI]
12. Frédéric Derbré, "Inactivity-induced oxidative stress: A central role in age-related sarcopenia?", European Journal of Sport Science, 14(sup1), pp.S98~S108 (2014), Informa UK Limited[DOI]
13. Catherine Moorwood, "Isometric and Eccentric Force Generation Assessment of Skeletal Muscles Isolated from Murine Models of Muscular Dystrophies", Journal of Visualized Experiments, (71), pp.e50036~ (2013), MyJove Corporation[DOI]
14. Mark Romanick, "Murine models of atrophy, cachexia, and sarcopenia in skeletal muscle", Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 1832(9), pp.1410~1420 (2013), Elsevier BV[DOI]
15. Guo An Yun, "Muscle mass, structural and functional investigations of senescence-accelerated mouse P8 (SAMP8)", Experimental Animals, 64(4), pp.425~433 (2015), Japanese Association for Laboratory Animal Science[DOI]
16. Takayuki Mito, "Transmitochondrial mito-miceΔ and mtDNA mutator mice, but not aged mice, share the same spectrum of musculoskeletal disorders", Biochem Biophys Res Commun, 456(4), pp.933~937 (2015), Elsevier BV[DOI]
17. Yuya Nakashima, "Preventive effects of Chlorella on skeletal muscle atrophy in muscle-specific mitochondrial aldehyde dehydrogenase 2 activity-deficient mice", BMC Complementary and Alternative Medicine, 14(1), pp.390~ (2014), Springer Science and Business Media LLC[DOI]
18. JONATHAN WANAGAT, "Mitochondrial DNA deletion mutations colocalize with segmental electron transport system abnormalities, muscle fiber atrophy, fiber splitting, and oxidative damage in sarcopenia", The FASEB Journal, 15(2), pp.322~332 (2001), Wiley[DOI]
19. M. Jane Mohler, "The Frailty Syndrome: Clinical measurements and basic underpinnings in humans and animals", Experimental Gerontology, 54, pp.6~13 (2014), Elsevier BV[DOI]
20. J. Walston, "The Physical and Biological Characterization of a Frail Mouse Model", The Journals of Gerontology Series A: Biological Sciences and Medical Sciences, 63(4), pp.391~398 (2008), Oxford University Press (OUP)[DOI]
21. Sarah M. Greising, "Skeletal muscle contractile function and neuromuscular performance in Zmpste24 −", − mice, a murine model of human progeria, 2012, pp.34(4), 805~819[DOI]
22. Guillermo Mariño, "Insulin-like growth factor 1 treatment extends longevity in a mouse model of human premature aging by restoring somatotroph axis function", Proceedings of the National Academy of Sciences, 107(37), pp.16268~16273 (2010), Proceedings of the National Academy of Sciences[DOI]
23. Sandrine Pereira, "HGPS and related premature aging disorders: From genomic identification to the first therapeutic approaches", Mechanisms of Ageing and Development, 129(7-8), pp.449~459 (2008), Elsevier BV[DOI]
24. Fabrizio d'Adda di Fagagna, "Effects of DNA nonhomologous end-joining factors on telomere length and chromosomal stability in mammalian cells", Current Biology, 11(15), pp.1192~1196 (2001), Elsevier BV[DOI]
25. Nathalie Didier, "Loss of a single allele for Ku80 leads to progenitor dysfunction and accelerated aging in skeletal muscle", EMBO Molecular Medicine, 4(9), pp.910~923 (2012), EMBO[DOI]
26. André Nussenzweig, "Requirement for Ku80 in growth and immunoglobulin V(D)J recombination", Nature, 382(6591), pp.551~555 (1996), Springer Science and Business Media LLC[DOI]
27. Aleksandra Trifunovic, "Premature ageing in mice expressing defective mitochondrial DNA polymerase", Nature, 429(6990), pp.417~423 (2004), Springer Science and Business Media LLC[DOI]
28. G. C. Kujoth, "Mitochondrial DNA Mutations, Oxidative Stress, and Apoptosis in Mammalian Aging", Science, 309(5733), pp.481~484 (2005), American Association for the Advancement of Science (AAAS)[DOI]
29. Joanna Groden, "Identification and characterization of the familial adenomatous polyposis coli gene", Cell, 66(3), pp.589~600 (1991), Elsevier BV[DOI]
30. Amy Rapaich Moser, "A Dominant Mutation That Predisposes to Multiple Intestinal Neoplasia in the Mouse", Science, 247(4940), pp.322~324 (1990), American Association for the Advancement of Science (AAAS)[DOI]
31. Kristen A. Mehl, "Myofiber degeneration", regeneration is induced in the cachecticApcMin, Journal of Applied Physiology, pp.2005, 99(6)[DOI]
32. Kristen A. Baltgalvis, "Interleukin-6 and cachexia inApcMin", +mice, 2008, pp.294(2), R393~R401[DOI]
33. Jinhua Shen, "Deficiency of MIP", MTMR14 phosphatase induces a muscle disorder by disrupting Ca2+ homeostasis, 2009, pp.11(6), 769~776[DOI]
34. Sandra Romero-Suarez, "Muscle-specific inositide phosphatase (MIP", MTMR14) is reduced with age and its loss accelerates skeletal muscle aging process by altering calcium homeostasis, 2010, pp.2(8), 504~513[DOI]
35. John J McCarthy, "Inducible Cre transgenic mouse strain for skeletal muscle-specific gene targeting", Skeletal Muscle, 2(1), pp.8~ (2012), Springer Science and Business Media LLC[DOI]
36. Alessandro Fanzani, "Molecular and cellular mechanisms of skeletal muscle atrophy: an update", Journal of Cachexia, Sarcopenia and Muscle, 3(3), pp.163~179 (2012), Wiley[DOI]
37. T. Lang, "Sarcopenia: etiology, clinical consequences, intervention, and assessment", Osteoporosis International, 21(4), pp.543~559 (2010), Springer Science and Business Media LLC[DOI]
38. Kunihiro Sakuma, "Sarcopenia and cachexia: the adaptations of negative regulators of skeletal muscle mass", Journal of Cachexia, Sarcopenia and Muscle, 3(2), pp.77~94 (2012), Wiley[DOI]
39. David J. Glass, "Skeletal muscle hypertrophy and atrophy signaling pathways", The International Journal of Biochemistry & Cell Biology, 37(10), pp.1974~1984 (2005), Elsevier BV[DOI]
40. Thea Shavlakadze, "Of bears, frogs, meat, mice and men: complexity of factors affecting skeletal muscle mass and fat", BioEssays, 28(10), pp.994~1009 (2006), Wiley[DOI]
41. Christian Rommel, "Mediation of IGF-1-induced skeletal myotube hypertrophy by PI(3)K", Akt, Akt, pp.GSK3 pathways, Nature Cell Biology[DOI]
42. Igor Vivanco, "The phosphatidylinositol 3-Kinase–AKT pathway in human cancer", Nature Reviews Cancer, 2(7), pp.489~501 (2002), Springer Science and Business Media LLC[DOI]
43. Giorgia Pallafacchina, "A protein kinase B-dependent and rapamycin-sensitive pathway controls skeletal muscle growth but not fiber type specification", Proceedings of the National Academy of Sciences, 99(14), pp.9213~9218 (2002), Proceedings of the National Academy of Sciences[DOI]
44. Stephan Wullschleger, "TOR Signaling in Growth and Metabolism", Cell, 124(3), pp.471~484 (2006), Elsevier BV[DOI]
45. Sue C. Bodine, "Akt", mTOR pathway is a crucial regulator of skeletal muscle hypertrophy and can prevent muscle atrophy in vivo, 2001, pp.3(11), 1014~1019[DOI]
46. Marco Sandri, "Foxo Transcription Factors Induce the Atrophy-Related Ubiquitin Ligase Atrogin-1 and Cause Skeletal Muscle Atrophy", Cell, 117(3), pp.399~412 (2004), Elsevier BV[DOI]
47. Jinghui Zhao, "FoxO3 Coordinately Activates Protein Degradation by the Autophagic", Lysosomal and Proteasomal Pathways in Atrophying Muscle Cells, 2007, pp.6(6), 472~483[DOI]
48. William S. Chen, "Growth retardation and increased apoptosis in mice with homozygous disruption of the akt1 gene", Genes & Development, 15(17), pp.2203~2208 (2001), Cold Spring Harbor Laboratory[DOI]
49. Han Cho, "Insulin Resistance and a Diabetes Mellitus-Like Syndrome in Mice Lacking the Protein Kinase Akt2 (PKBβ)", Science, 292(5522), pp.1728~1731 (2001), American Association for the Advancement of Science (AAAS)[DOI]
50. Marcus D. Goncalves, "Akt Deficiency Attenuates Muscle Size and Function but Not the Response to ActRIIB Inhibition", PLoS ONE, 5(9), pp.e12707~ (2010), Public Library of Science (PLoS)[DOI]
51. Ji Luo, "Class IA phosphoinositide 3-kinase regulates heart size and physiological cardiac hypertrophy", Molecular and Cellular Biology, 25(21), pp.9491~9502 (2005), American Society for Microbiology[DOI]
52. Goldberg AL, "Mechanism of work-induced hypertrophy of skeletal muscle", Med Sci Sports, 7, pp.185~198 (1975)
53. Craig A. Goodman, "The role of skeletal muscle mTOR in the regulation of mechanical load-induced growth", The Journal of Physiology, 589(22), pp.5485~5501 (2011), Wiley[DOI]
54. Troy A. Hornberger, "Mechanotransduction and the regulation of mTORC1 signaling in skeletal muscle", The International Journal of Biochemistry & Cell Biology, 43(9), pp.1267~1276 (2011), Elsevier BV[DOI]
55. Ana M. Fernández, "Functional inactivation of the IGF-I and insulin receptors in skeletal muscle causes type 2 diabetes", Genes & Development, 15(15), pp.1926~1934 (2001), Cold Spring Harbor Laboratory[DOI]
56. Espen E. Spangenburg, "A functional insulin-like growth factor receptor is not necessary for load-induced skeletal muscle hypertrophy", The Journal of Physiology, 586(1), pp.283~291 (2008), Wiley[DOI]
57. Dongsheng Cai, "IKKβ", NF-κB Activation Causes Severe Muscle Wasting in Mice, 2004, pp.119(2), 285~298[DOI]
58. R. Bridge Hunter, "Activation of an alternative NF-kappaB pathway in skeletal muscle during disuse atrophy", The FASEB Journal, 16(6), pp.529~538 (2002), Wiley[DOI]
59. A. H. V. Remels, "TNF-α impairs regulation of muscle oxidative phenotype: implications for cachexia?", The FASEB Journal, 24(12), pp.5052~5062 (2010), Wiley[DOI]
60. Ashley Wysong, "NF-κB Inhibition Protects against Tumor-Induced Cardiac Atrophy in Vivo", The American Journal of Pathology, 178(3), pp.1059~1068 (2011), Elsevier BV[DOI]
61. Marina Bar-Shai, "The effect of hindlimb immobilization on acid phosphatase, metalloproteinases and nuclear factor-κB in muscles of young and old rats", Mechanisms of Ageing and Development, 126(2), pp.289~297 (2005), Elsevier BV[DOI]
62. Tracey Phillips, "Muscle fiber‐specific apoptosis and TNF‐α signaling in sarcopenia are attenuated by life‐long calorie restriction", The FASEB Journal, 19(6), pp.1~33 (2005), Wiley[DOI]
63. Se-Jin Lee, "REGULATION OF MUSCLE MASS BY MYOSTATIN", Annual Review of Cell and Developmental Biology, 20(1), pp.61~86 (2004), Annual Reviews[DOI]
64. Alexandra C. McPherron, "Regulation of skeletal muscle mass in mice by a new TGF-p superfamily member", Nature, 387(6628), pp.83~90 (1997), Springer Science and Business Media LLC[DOI]
65. David L. Allen, "Regulation of myostatin expression and myoblast differentiation by FoxO and SMAD transcription factors", American Journal of Physiology-Cell Physiology, 292(1), pp.C188~C199 (2007), American Physiological Society[DOI]
66. Anne Ulrike Trendelenburg, "Myostatin reduces Akt", TORC1, American Journal of Physiology-Cell Physiology, pp.2009, 296(6)[DOI]
67. Victoria Siriett, "Prolonged absence of myostatin reduces sarcopenia", Journal of Cellular Physiology, 209(3), pp.866~873 (2006), Wiley[DOI]
68. N. K. LeBrasseur, "Myostatin Inhibition Enhances the Effects of Exercise on Performance and Metabolic Outcomes in Aged Mice", The Journals of Gerontology Series A: Biological Sciences and Medical Sciences, 64A(9), pp.940~948 (2009), Oxford University Press (OUP)[DOI]
69. Ilham Chelh, "Molecular profiles of Quadriceps muscle in myostatin-null mice reveal PI3K and apoptotic pathways as myostatin targets", BMC Genomics, 10(1), pp.196~ (2009), Springer Science and Business Media LLC[DOI]
70. Christopher D. McMahon, "Myostatin-deficient mice lose more skeletal muscle mass than wild-type controls during hindlimb suspension", American Journal of Physiology-Endocrinology and Metabolism, 285(1), pp.E82~E87 (2003), American Physiological Society[DOI]
71. Fadia Haddad, "Aging-sensitive cellular and molecular mechanisms associated with skeletal muscle hypertrophy", Journal of Applied Physiology, 100(4), pp.1188~1203 (2006), American Physiological Society[DOI]
72. Michael R. Morissette, "Effects of myostatin deletion in aging mice", Aging Cell, 8(5), pp.573~583 (2009), Wiley[DOI]
73. Ning Chen, "Role and regulation of autophagy in cancer", Biochimica et Biophysica Acta (BBA) - Molecular Cell Research, 1793(9), pp.1516~1523 (2009), Elsevier BV[DOI]
74. Daniel J. Klionsky, "Autophagy: from phenomenology to molecular understanding in less than a decade", Nature Reviews Molecular Cell Biology, 8(11), pp.931~937 (2007), Springer Science and Business Media LLC[DOI]
75. Goran Petrovski, "Does autophagy take a front seat in lifespan extension?", Journal of Cellular and Molecular Medicine, 14(11), pp.2543~2551 (2010), Wiley[DOI]
76. Gabriella Dobrowolny, "Skeletal Muscle Is a Primary Target of SOD1G93A-Mediated Toxicity", Cell Metabolism, 8(5), pp.425~436 (2008), Elsevier BV[DOI]
77. Michela Aucello, "Localized accumulation of oxidative stress causes muscle atrophy through activation of an autophagic pathway", Autophagy, 5(4), pp.527~529 (2009), Informa UK Limited[DOI]
78. Stefano Schiaffino, "Studies on the effect of denervation in developing muscle. II. The lysosomal system", Journal of Ultrastructure Research, 39(1-2), pp.1~14 (1972), Elsevier BV[DOI]
79. Noboru Mizushima, "In Vivo Analysis of Autophagy in Response to Nutrient Starvation Using Transgenic Mice Expressing a Fluorescent Autophagosome Marker", Molecular Biology of the Cell, 15(3), pp.1101~1111 (2004), American Society for Cell Biology (ASCB)[DOI]
80. Zhifen Yang, "Mammalian autophagy: core molecular machinery and signaling regulation", Current Opinion in Cell Biology, 22(2), pp.124~131 (2010), Elsevier BV[DOI]
81. Cristina Mammucari, "FoxO3 Controls Autophagy in Skeletal Muscle In Vivo", Cell Metabolism, 6(6), pp.458~471 (2007), Elsevier BV[DOI]
82. Yasutomi Kamei, "Skeletal muscle FOXO1 (FKHR) transgenic mice have less skeletal muscle mass, down-regulated Type I (slow twitch", red muscle) fiber genes, and impaired glycemic control, 2004, pp.279(39), 41114~41123[DOI]
83. Tadahiro Kitamura, "A Foxo", Notch pathway controls myogenic differentiation and fiber type specification, 2007, pp.117(9), 2477~2485[DOI]
84. Valérie Risson, "Muscle inactivation of mTOR causes metabolic and dystrophin defects leading to severe myopathy", Journal of Cell Biology, 187(6), pp.859~874 (2009), Rockefeller University Press[DOI]
85. Sue C. Bodine, "Identification of Ubiquitin Ligases Required for Skeletal Muscle Atrophy", Science, 294(5547), pp.1704~1708 (2001), American Association for the Advancement of Science (AAAS)[DOI]
86. Christian C Witt, "Cooperative control of striated muscle mass and metabolism by MuRF1 and MuRF2", The EMBO Journal, 27(2), pp.350~360 (2008), Wiley[DOI]

관련 논문AI추천