Acute Cerebral Infarction in Carbon Monoxide Poisoning
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초록
Hypoxic brain damage after carbon monoxide (CO) occurs predominantly in the basal ganglia, cerebral white matter and cerebral cortex. CO poisoning is complicated by various neurological manifestations as well as hemorrhagic infarction. Acute cerebral thromboembolic infarction following CO poisoning is very rare. We experienced a case of thromboembolic infarction in the middle cerebral artery territory occurred in a healthy young adult without any risk factor for stroke during treatment for hypoxic encephalopathy caused by acute CO poisoning. (Korean J Stroke 2011;13:92-95)
Abstract
Hypoxic brain damage after carbon monoxide (CO) occurs predominantly in the basal ganglia, cerebral white matter and cerebral cortex. CO poisoning is complicated by various neurological manifestations as well as hemorrhagic infarction. Acute cerebral thromboembolic infarction following CO poisoning is very rare. We experienced a case of thromboembolic infarction in the middle cerebral artery territory occurred in a healthy young adult without any risk factor for stroke during treatment for hypoxic encephalopathy caused by acute CO poisoning. (Korean J Stroke 2011;13:92-95)
참고문헌 (8)
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