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A Novel Case of Extreme Thrombocytosis in Acute Myeloid Leukemia Associated With Isochromosome 17q and Copy Neutral Loss of Heterozygosity

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참고문헌 (9)

  1. Fioretos T, "Isochromosome 17q in blast crisis of chronic myeloid leukemia and in other hematologic malignancies is the result of clustered breakpoints in 17p11 and is not associated with coding TP53 mutations", Blood, 94, pp.225~232 (1999)
  2. Mertens F, "Isochromosomes in neoplasia", Genes Chromosomes Cancer, 10, pp.221~230 (1994)
  3. Kanagal-Shamanna R, "Myeloid neoplasms with isolated isochromosome17q represent a clinicopathologic entity associated with myelodysplastic", myeloproliferative features, a high risk of leukemic transformation, and wild-type TP53, 2012, pp.118, 2879~2888
  4. Lim G, "Acute myeloid leukemia associated with t(1;3)(p36;q21)and extreme thrombocytosis : a clinical study with literature review", Cancer Genet Cytogenet, 203, pp.187~192 (2010)
  5. "t(1;3)(p36;q21). Atlas Genet Cytogenet Oncol Haematol" (2002)[URL]
  6. Simmers RN, "Localization of the human G-CSF gene to the region of a breakpoint in the translocation typical of acute promyelocytic leukemia", Hum Genet, 78, pp.134~136 (1988)
  7. Chang KS, "The localization of the human myeloperoxydase gene is in close proximity to the translocation breakpoint in acute promyelocytic leukemia", Leukemia, 1, pp.458~462 (1987)
  8. Fiedler W, "GCSF gene is expressed but not rearranged in a patient with isochromosome 17q positive acute nonlymphocytic leukemia", Cancer Genet Cytogenet, 68, pp.49~51 (1993)
  9. Jerez A, "Loss of heterozygosity in 7q myeloid disorders : clinical associations and genomic pathogenesis", Blood, 119, pp.6109~6117 (2012)

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