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First Case of Double T-Cell Receptor Alpha/Delta Rearrangements of t(11;14) and inv(14) and Subsequent JAK2 Rearrangement in a Patient With T-cell Acute Lymphoblastic Leukemia

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참고문헌 (10)

  1. Belver L, "The genetics and mechanisms of T cell acute lymphoblastic leukaemia", Nat Rev Cancer, 16, pp.494~507 (2016)
  2. Chen B, "Identification of fusion genes and characterization of transcriptome features in T-cell acute lymphoblastic leukemia", Proc Natl Acad Sci U S A, 115, pp.373~378 (2018)
  3. Reiter A, "The t(8;9)(p22;p24) is a recurrent abnormality in chronic and acute leukemia that fuses PCM1 to JAK2", Cancer Res, 65, pp.2662~2667 (2005)
  4. Murati A, "PCM1-JAK2 fusion in myeloproliferative disorders and acute erythroid leukemia with t(8;9) translocation", Leukemia, 19, pp.1692~1696 (2005)
  5. Liu Y, "The genomic landscape of pediatric and young adult T-lineage acute lymphoblastic leukemia", Nat Genet, 49, pp.1211~1218 (2017)
  6. Wilkinson A, "Inversion(14)(q11q32)in a patient with childhood T-cell acute lymphoblastic leukemia", Cancer Genet Cytogenet, 88, pp.76~79 (1996)
  7. Przybylski GK, "Disruption of the BCL11B gene through inv(14)(q11.2q32.31) results in the expression of BCL11B-TRDC fusion transcripts and is associated with the absence of wild-type BCL11B transcripts in T-ALL", Leukemia, 19, pp.201~208 (2005)
  8. Swerdlow SH, "WHO classification of tumours of haematopoietic and lymphoid tissues", pp.78~79 (2017), International Agency for Research on Cancer
  9. Tang G, "Hematopoietic neoplasms with 9p24", JAK2 rearrangement : a multicenter study, 2019, pp.32, 490~498
  10. Girardi T, "The genetics and molecular biology of T-ALL", Blood, 129, pp.1113~1123 (2017)

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