BLT2, a leukotriene B4 receptor 2, as a novel prognostic biomarker of triple-negative breast cancer

키워드

참고문헌 (36)

1. Anders CK, "Poly(ADP-Ribose) polymerase inhibition: "targeted" therapy for triple-negative breast cancer", Clin Cancer Res, 16, pp.4702~4710 (2010)[DOI]
2. Carey L, "Triple-negative breast cancer: disease entity or title of convenience?", Nat Rev Clin Oncol, 7, pp.683~692 (2010)[DOI]
3. Kassam F, "Survival outcomes for patients with metastatic triple-negative breast cancer: implications for clinical practice and trial design", Clin Breast Cancer, 9, pp.29~33 (2009)[DOI]
4. Dinarello CA, "The paradox of pro-inflammatory cytokines in cancer", Cancer Metastasis Rev, 25, pp.307~313 (2006)[DOI]
5. Tager AM, "BLT1 and BLT2: the leukotriene B(4) receptors", Prostaglandins Leukot Essent Fatty Acids, 69, pp.123~134 (2003)[DOI]
6. Park GS, "Myeloid differentiation primary response gene 88-leukotriene B4 receptor 2 cascade mediates lipopolysaccharide-potentiated invasiveness of breast cancer cells", Oncotarget, 6, pp.5749~5759 (2015)
7. Maiello MR, "EGFR and MEK Blockade in Triple Negative Breast Cancer Cells", J Cell Biochem, 116, pp.2778~2785 (2015)[DOI]
8. Lonne GK, "Protein kinase Cdelta supports survival of MDA-MB-231 breast cancer cells by suppressing the ERK1", 2 pathway, 2009, pp.284, 33456~33465
9. Bogucki DJ, "Numerical study of light scattering by a boundary-layer flow", Appl Opt, 44, pp.5286~5291 (2005)[DOI]
10. Weng MS, "The interplay of reactive oxygen species and the epidermal growth factor receptor in tumor progression and drug resistance", J Exp Clin Cancer Res, 37, pp.61~ (2018)[DOI]
11. Pelicano H, "Mitochondrial dysfunction in some triple-negative breast cancer cell lines: role of mTOR pathway and therapeutic potential", Breast Cancer Res, 16, pp.434~ (2014)[DOI]
12. Puca R, "Nox1 is involved in p53 deacetylation and suppression of its transcriptional activity and apoptosis", Free Radic Biol Med, 48, pp.1338~1346 (2010)[DOI]
13. Choi JA, "Pro-survival of estrogen receptor-negative breast cancer cells is regulated by a BLT2-reactive oxygen species-linked signaling pathway", Carcinogenesis, 31, pp.543~551 (2010)[DOI]
14. Blot E, "Cooperation between monocytes and breast cancer cells promotes factors involved in cancer aggressiveness", Br J Cancer, 88, pp.1207~1212 (2003)[DOI]
15. Chittezhath M, "Molecular profiling reveals a tumor-promoting phenotype of monocytes and macrophages in human cancer progression", Immunity, 41, pp.815~829 (2014)[DOI]
16. Nakasone ES, "Harnessing the immune system in the battle against breast cancer", Drugs Context, 7, pp.212520~ (2018)
17. Brown EJ, "Adhesive interactions in the immune system", Trends Cell Biol, 7, pp.289~295 (1997)[DOI]
18. Ohene-Abuakwa Y, "Adhesion molecules as diagnostic tools in tumor pathology", Int J Surg Pathol, 8, pp.191~200 (2000)[DOI]
19. Zhong L, "Endothelial microRNAs regulating the NF-kappaB pathway and cell adhesion molecules during inflammation", FASEB J (2018)
20. Hubbard AK, "Intercellular adhesion molecule-1 (ICAM-1) expression and cell signaling cascades", Free Radic Biol Med, 28, pp.1379~1386 (2000)[DOI]
21. Evani SJ, "Monocytes mediate metastatic breast tumor cell adhesion to endothelium under flow", FASEB J, 27, pp.3017~3029 (2013)[DOI]
22. Rosette C, "Role of ICAM1 in invasion of human breast cancer cells", Carcinogenesis, 26, pp.943~950 (2005)[DOI]
23. Strell C, "Neutrophil granulocytes promote the migratory activity of MDA-MB-468 human breast carcinoma cells via ICAM-1", Exp Cell Res, 316, pp.138~148 (2010)[DOI]
24. Schroder C, "Prognostic value of intercellular adhesion molecule (ICAM)-1 expression in breast cancer", J Cancer Res Clin Oncol, 137, pp.1193~1201 (2011)[DOI]
25. 박근수, "LPS Up-Regulates ICAM-1 Expression in Breast Cancer Cells by Stimulating a MyD88-BLT2-ERKLinked Cascade, Which Promotes Adhesion to Monocytes", Molecules and Cells, 38(9), pp.821~828 (2015), 한국분자세포생물학회[DOI]
26. Neophytou C, "Molecular mechanisms and emerging therapeutic targets of triple-negative breast cancer metastasis", Front Oncol, 8, pp.31~ (2018)[DOI]
27. Christofori G, "New signals from the invasive front", Nature, 441, pp.444~450 (2006)[DOI]
28. Liang Z, "Downregulation of microRNA-206 promotes invasion and angiogenesis of triple negative breast cancer", Biochem Biophys Res Commun, 477, pp.461~466 (2016)[DOI]
29. Ma Y, "IL-6, IL-8 and TNF-alpha levels correlate with disease stage in breast cancer patients", Adv Clin Exp Med, 26, pp.421~426 (2017)[DOI]
30. Hartman ZC, "Growth of triple-negative breast cancer cells relies upon coordinate autocrine expression of the proinflammatory cytokines IL-6 and IL-8", Cancer Res, 73, pp.3470~3480 (2013)[DOI]
31. De Larco JE, "A potential role for interleukin-8 in the metastatic phenotype of breast carcinoma cells", Am J Pathol, 158, pp.639~646 (2001)[DOI]
32. Kim H, "BLT2 up-regulates interleukin-8 production and promotes the invasiveness of breast cancer cells", PLoS One, 7, pp.e49186~ (2012)[DOI]
33. Madden SF, "BreastMark: an integrated approach to mining publicly available transcriptomic datasets relating to breast cancer outcome", Breast Cancer Res, 15, pp.R52~ (2013)[DOI]
34. Connolly C, "Expression of anaesthetic and analgesic drug target genes in excised breast tumour tissue: Association with clinical disease recurrence or metastasis", PLoS One, 12, pp.e0177105~ (2017)[DOI]
35. Gao J, "Integrative analysis of complex cancer genomics and clinical profiles using the cBioPortal", Sci Signal, 6 (2013)
36. Lavigne AC, "Increased macroH2A1.1 expression correlates with poor survival of triple-negative breast cancer patients", PLoS One, 9, pp.e98930~ (2014)[DOI]

관련 논문AI추천