부분 무치증 환아의 증례보고
Oligodontia : Case report
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부분 무치증은 제 3대구치를 제외한 6개 이상의 선천적 치아 결손으로 정의된다. 제3대구치를 제외한 선천적 치아 결손의 유병율은 1.6.9.6%이며 부분 무치증의 유병율은 0.08.1.1%이다. 가장 많이 이환되는 치아는 하악 제 2소구치이며 그 다음은 상악 측절치, 상악 제2소구치 순으로 호발하며 영구치에 비해 유치에서는 드물고 여성에게 호발하는 경향이 있다. 부분 무치증은 외배엽 이형성증과 같은 특정 증후군과 연관되거나 치판의 생리적인 장애나 파열, 공간적 제한, 치성 상피의 기능적인 비정상, 기저 간엽세포의 유도 실패나 유전적 영향 등의 원인에서 유래된다고 알려져 있다. 부분 무치증은 안모의 심미적 문제와 함께 치아의 교합 이상 등 기능적인 장애가 야기될 수 있으므로 임상 검사와 방사선 검사를 통한 조기 진단이 필요하며, 이에 따른 적절한 치료계획이 요구된다. 이에 본 증례에서는 특별한 전신질환이 없는 부분 무치증 환아의 구강내 소견 및치료경과에 대해 보고하고자 한다.
Abstract
Oligodontia is defined as the congenital absence of six or more teeth in dentition, excluding the third molars. The prevalence of congenital missing teeth is about 1.6 to 9.6% of population and the prevalence of oligidontia is about 0.08 to 1.1%. The mandibular second premolar is the most frequently absent after the third molar, followed by the maxillary lateral incisor and upper second premolar. Females seem to be affected slightly more than males. Oligodontia may occur either in isolation, or as a part of a syndrome such as ectodermal dysplasia. Different causes are possible for oligodontia: physical obstruction or distruction of the dental lamina, space limitation, functional abnormalities of the dental epithelium, failure of induction of the underlying mesenchyme, chemotherapy, radiotherapy or genetic factor. Because oligodontia would result in esthetic and functional problems, such as facial asymmetry or occlusal disharmony, early diagnosis from clinical and radiographic examination was necessary. And appropriate treatment plan should be followed. This case report was about oral conditions and treatment of the oligodontia patients who have no specific systemic disease.
참고문헌 (30)
- 정혜선, "가족력을 동반한 부분적 무치증에 대한 증례보고", 24, pp.179~185 (1997)
- Anita F, "Hypodontia in orthodontically treated children", 27, pp.457~460 (2005)
- 박상억, "다수 치아의 선천성 결손", pp.115~127 (1992)
- Silva Meza R, "Radiographic assessment of congenitally missing teeth in orthodontic patients", 13, pp.112~116 (2003)
- Tsai PF, "Oligodontia. Case report", 29, pp.191~193 (1998)
- 이재영, "가족력에 의한 선천성 치아결손증", pp.127~134 (1988)
- Granath L, "Numerical variations in primary dentition and their correlation with the permanent dentition", pp.348~357 (1961)
- Ravn JJ, "supernumerary teeth and fused teeth in the primary dentition", pp.1~6 (1971)
- Scand J Dent Res, "The prevalence of malocclusion in Swedish schoolchildren", pp.12~21 (1973)
- Vastardis H, "The genetics of human tooth agenesis: New discoveries for understanding dental anomalies", 117, pp.650~656 (2000)
- Jorgenson RJ, "Clinician’s view of hypodontia", pp.283~286 (1980)
- Schalk-van der Weide Y, "Distribution of missing teeth and tooth morphology in patients with oligodontia", pp.133~140 (1992)
- Goldenberg M, "Clinical, radiographic, and genetic evaluation of a novel form of autosomal-dominant oligodontia", 79, pp.1469~1475 (2000)
- Rolling S, "Oligodontia in Danish schoolchildren", 59, pp.111~112 (2001)
- Nordgarden H, "Reported prevalence of congenitally missing teeth in two Norwegian countries", 19, pp.258~261 (2002)
- Bergendal B, "Signs and symptoms from ectodermal organs in young Swedish individuals with oligodontia", 16, pp.320~326 (2006)
- Endo T, "A survey of hypodontia in Japanese orthodontic patients", 129, pp.29~35 (2006)
- Fujiwara T, "Simultaneous occurrence of unusual odontodysplasia and oligodontia in the permanent dentition: report of a case", 10, pp.341~347 (2000)
- Clayton JM, "Congenital dental anormalies occuring in 3", pp.557~ (1956)
- Witkop CJ Jr, "Hereditary defects of dentin", pp.25~45 (1975)
- Berlin, "quoted in 9" (1922)
- Rushton MA, "A case of dentinal dysplasia", pp.369~373 (1939)
- Shields ED, "A proposed classification for heritable human dentine defects with a description of a new entity", pp.543~53 (1973)
- Bahn SL, "Radiographic manifestations of an unusual combination Types I and Type II dentin dysplasia", pp.317~22 (1978)
- Shankly PE, "Dentinal dysplasia type I: report of a case", 9, pp.37~42 (1999)
- Gerits A, "Exclusion of coding region mutations in MSX1, PAX9 and AXIN2 in eight patients with severe oligodontia phenotype", 9, pp.129~136 (2006)
- Ogawa T, "Studies on Pax9-Msx1 protein interactions", 50, pp.141~145 (2005)
- Lammi L, "Mutations in AXIN2 cause familial tooth agenesis and predispose to colorectal cancer", 74, pp.1043~1051 (2004)
- Scarel RM, "Absence of mutations in the homeodomain of the MSX1 gene in patients with hypodontia", 92, pp.346~349 (2000)
- Frazier-Bowers SA, "A unique form of hypodontia seen in Vietnamese patients: clinical and molecular analysis", 40, pp.79~83 (2003)