Origin of Osteoclasts: Osteoclast Precursor Cells

참고문헌 (101)

1. Tsukasaki M, "Osteoimmunology : Evolving concepts in bone-immune interactions in health and disease", Nat Rev Immunol, 19, pp.626~642 (2019)[DOI]
2. Park-Min KH, "Mechanisms involved in normal and pathological osteoclastogenesis", Cell Mol Life Sci, 75, pp.2519~2528 (2018)[DOI]
3. Novack DV, "The osteoclast : friend or foe?", Annu Rev Pathol, 3, pp.457~484 (2008)[DOI]
4. Schett G, "Bone erosion in rheumatoid arthritis: mechanisms, diagnosis and treatment", Nat Rev Rheumatol, 8, pp.656~664 (2012)[DOI]
5. Walker DG, "Bone resorption restored in osteopetrotic mice by transplants of normal bone marrow and spleen cells", Science, 190, pp.784~785 (1975)[DOI]
6. Teti A, "Early treatment of osteopetrosis : Paradigm shift to marrow cell transplantation", Bone, 164, pp.116512~ (2022)[DOI]
7. Johansson MK, "Hematopoietic stem cell-targeted neonatal gene therapy reverses lethally progressive osteopetrosis in oc", oc mice, 2007, pp.109, 5178~5185
8. Kurihara N, "Identification of committed mononuclear precursors for osteoclast-like cells formed in long term human marrow cultures", Endocrinology, 126, pp.2733~2741 (1990)[DOI]
9. Udagawa N, "Origin of osteoclasts : Mature monocytes and macrophages are capable of differentiating into osteoclasts under a suitable microenvironment prepared by bone marrow-derived stromal cells", Proc Natl Acad Sci U S A, 87, pp.7260~7264 (1990)[DOI]
10. Matsuzaki K, "Osteoclast differentiation factor(ODF)induces osteoclast-like cell formation in human peripheral blood mononuclear cell cultures", Biochem Biophys Res Commun, 246, pp.199~204 (1998)[DOI]
11. Hattersley G, "Identification of osteoclast precursors in multilineage hemopoietic colonies", Endocrinology, 128, pp.259~262 (1991)[DOI]
12. Park-Min KH, "Osteoclasts : Other functions", Bone, 165, pp.116576~ (2022)[DOI]
13. Geissmann F, "Development of monocytes, macrophages, and dendritic cells", Science, 327, pp.656~661 (2010)[DOI]
14. Hettinger J, "Origin of monocytes and macrophages in a committed progenitor", Nat Immunol, 14, pp.821~830 (2013)[DOI]
15. Guilliams M, "Developmental and functional heterogeneity of monocytes", Immunity, 49, pp.595~613 (2018)[DOI]
16. Shi C, "Monocyte recruitment during infection and inflammation", Nat Rev Immunol, 11, pp.762~774 (2011)[DOI]
17. Ginhoux F, "Monocytes and macrophages : developmental pathways and tissue homeostasis", Nat Rev Immunol, 14, pp.392~404 (2014)[DOI]
18. Rivollier A, "Immature dendritic cell transdifferentiation into osteoclasts : a novel pathway sustained by the rheumatoid arthritis microenvironment", Blood, 104, pp.4029~4037 (2004)[DOI]
19. Speziani C, "Murine dendritic cell transdifferentiation into osteoclasts is differentially regulated by innate and adaptive cytokines", Eur J Immunol, 37, pp.747~757 (2007)[DOI]
20. Gallois A, "Genome-wide expression analyses establish dendritic cells as a new osteoclast precursor able to generate bone-resorbing cells more efficiently than monocytes", J Bone Miner Res, 25, pp.661~672 (2010)[DOI]
21. Murphy TL, "Dendritic cells in cancer immunology", Cell Mol Immunol, 19, pp.3~13 (2022)[DOI]
22. Yosef N, "The phenotypic and functional properties of mouse yolk-sac-derived embryonic macrophages", Dev Biol, 442, pp.138~154 (2018)[DOI]
23. Grzybowski A, "Re: Bhende et al.: Risk factors for endophthalmitis after pars plana vitrectomies in a tertiary eye institute in India (Ophthalmol Retina. 2018 doi:10.1016", j.oret.2018.01.001), 2018, pp.2, e7~
24. Schulz C, "A lineage of myeloid cells independent of Myb and hematopoietic stem cells", Science, 336, pp.86~90 (2012)[DOI]
25. Bazan JF, "A new class of membrane-bound chemokine with a CX3C motif", Nature, 385, pp.640~644 (1997)[DOI]
26. Meghraoui-Kheddar A, "Revising CX3CR1 expression on murine classical and non-classical monocytes", Front Immunol, 11, pp.1117~ (2020)[DOI]
27. Lichanska AM, "Origins and functions of phagocytes in the embryo", Exp Hematol, 28, pp.601~611 (2000)[DOI]
28. Yahara Y, "The origins and roles of osteoclasts in bone development, homeostasis and repair", Development, 149, pp.dev199908~ (2022)[DOI]
29. Yahara Y, "Erythromyeloid progenitors give rise to a population of osteoclasts that contribute to bone homeostasis and repair", Nat Cell Biol, 22, pp.49~59 (2020)[DOI]
30. Feehan J, "Circulating osteogenic precursor cells : Building bone from blood", EBioMedicine, 39, pp.603~611 (2019)[DOI]
31. Jacome-Galarza CE, "Identification, characterization, and isolation of a common progenitor for osteoclasts, macrophages, and dendritic cells from murine bone marrow and periphery", J Bone Miner Res, 28, pp.1203~1213 (2013)[DOI]
32. Wu CC, "Diagnosis and management of osteopetrosis : Consensus gidelines from the osteopetrosis working group", J Clin Endocrinol Metab, 102, pp.3111~3123 (2017)[DOI]
33. Stepensky P, "Stem cell transplantation for osteopetrosis in patients beyond the age of 5 years", Blood Adv, 3, pp.862~868 (2019)[DOI]
34. Göthlin G, "On the histogenesis of the cells in fracture callus. Electron microscopic autoradiographic observations in parabiotic rats and studies on labeled monocytes", Virchows Arch B Cell Pathol, 12, pp.318~329 (1973)
35. Novak S, "Osteoclasts derive predominantly from bone marrow-resident CX(3)CR1(+) precursor cells in homeostasis, whereas circulating CX(3)CR1(+) cells contribute to osteoclast development during fracture repair", J Immunol, 204, pp.868~878 (2020)[DOI]
36. Jacome-Galarza CE, "Developmental origin, functional maintenance and genetic rescue of osteoclasts", Nature, 568, pp.541~545 (2019)[DOI]
37. Pronk CJ, "Elucidation of the phenotypic, functional, and molecular topography of a myeloerythroid progenitor cell hierarchy", Cell Stem Cell, 1, pp.428~442 (2007)[DOI]
38. Seita J, "Hematopoietic stem cell : Self-renewal versus differentiation", Wiley Interdiscip Rev Syst Biol Med, 2, pp.640~653 (2010)[DOI]
39. Arai F, "Commitment and differentiation of osteoclast precursor cells by the sequential expression of c-Fms and receptor activator of nuclear factor kappaB(RANK)receptors", J Exp Med, 190, pp.1741~1754 (1999)[DOI]
40. Xiao Y, "Macrophages and osteoclasts stem from a bipotent progenitor downstream of a macrophage", osteoclast, Blood Adv, pp.2017, 1[DOI]
41. Lee MY, "Identification and characterization of osteoclast progenitors by clonal analysis of hematopoietic cells", Blood, 80, pp.1710~1716 (1992)
42. Muguruma Y, "Isolation and characterization of murine clonogenic osteoclast progenitors by cell surface phenotype analysis", Blood, 91, pp.1272~1279 (1998)
43. Fogg DK, "A clonogenic bone marrow progenitor specific for macrophages and dendritic cells", Science, 311, pp.83~87 (2006)[DOI]
44. Katavić V, "The surface antigen CD45R identifies a population of estrogen-regulated murine marrow cells that contain osteoclast precursors", Bone, 32, pp.581~590 (2003)[DOI]
45. Jacquin C, "Identification of multiple osteoclast precursor populations in murine bone marrow", J Bone Miner Res, 21, pp.67~77 (2006)[DOI]
46. Charles JF, "Inflammatory arthritis increases mouse osteoclast precursors with myeloid suppressor function", J Clin Invest, 122, pp.4592~4605 (2012)[DOI]
47. Das A, "Monocyte subsets with high osteoclastogenic potential and their epigenetic regulation orchestrated by IRF8", J Bone Miner Res, 36, pp.199~214 (2021)[DOI]
48. Fujikawa Y, "The human osteoclast precursor circulates in the monocyte fraction", Endocrinology, 137, pp.4058~4060 (1996)[DOI]
49. Passlick B, "Identification and characterization of a novel monocyte subpopulation in human peripheral blood", Blood, 74, pp.2527~2534 (1989)
50. Ziegler-Heitbrock L, "Nomenclature of monocytes and dendritic cells in blood", Blood, 116, pp.e74~e80 (2010)[DOI]
51. Wong KL, "The three human monocyte subsets : Implications for health and disease", Immunol Res, 53, pp.41~57 (2012)[DOI]
52. Sánchez-Torres C, "CD16+ and CD16-human blood monocyte subsets differentiate in vitro to dendritic cells with different abilities to stimulate CD4+ T cells", Int Immunol, 13, pp.1571~1581 (2001)[DOI]
53. Ravenhill BJ, "Comprehensive cell surface proteomics defines markers of classical, intermediate and non-classical monocytes", Sci Rep, 10, pp.4560~ (2020)[DOI]
54. Cren M, "Differential accumulation and activation of monocyte and dendritic cell subsets in Inflamed synovial fluid discriminates between juvenile idiopathic arthritis and septic arthritis", Front Immunol, 11, pp.1716~ (2020)[DOI]
55. Sprangers S, "Different blood-borne human osteoclast precursors respond in distinct ways to IL-17A", J Cell Physiol, 231, pp.1249~1260 (2016)[DOI]
56. Chiu YG, "CD16(FcRgammaIII)as a potential marker of osteoclast precursors in psoriatic arthritis", Arthritis Res Ther, 12, pp.R14~ (2010)[DOI]
57. Damasceno D, "Monocyte subsets and serum inflammatory and bone-associated markers in monoclonal gammopathy of undetermined significance and multiple myeloma", Cancers(Basel), 13, pp.1454~ (2021)[DOI]
58. Xue J, "CD14(+)CD16(-)monocytes are the main precursors of osteoclasts in rheumatoid arthritis via expressing Tyro3TK", Arthritis Res Ther, 22, pp.221~ (2020)[DOI]
59. Lari R, "The proliferative human monocyte subpopulation contains osteoclast precursors", Arthritis Res Ther, 11, pp.R23~ (2009)[DOI]
60. Komano Y, "Identification of a human peripheral blood monocyte subset that differentiates into osteoclasts", Arthritis Res Ther, 8, pp.R152~ (2006)[DOI]
61. Flanagan AM, "Generating human osteoclasts in vitro from bone marrow and peripheral blood", Methods Mol Med, 80, pp.113~128 (2003)[DOI]
62. Takahashi N, "Osteoclast-like cells form in long-term human bone marrow but not in peripheral blood cultures", J Clin Invest, 83, pp.543~550 (1989)[DOI]
63. Hughes DE, "Inhibition of osteoclast-like cell formation by bisphosphonates in long-term cultures of human bone marrow", J Clin Invest, 83, pp.1930~1935 (1989)[DOI]
64. Heymann D, "Upmodulation of multinucleated cell formation in long-term human bone marrow cultures by leukaemia inhibitory factor(LIF)", Cytokine, 9, pp.46~52 (1997)[DOI]
65. Sarma U, "Macrophage colony-stimulating factor induces substantial osteoclast generation and bone resorption in human bone marrow cultures", Blood, 88, pp.2531~2540 (1996)
66. Xiao Y, "Identification of the common origins of osteoclasts, macrophages, and dendritic cells in human hematopoiesis", Stem Cell Reports, 4, pp.984~994 (2015)[DOI]
67. Doulatov S, "Hematopoiesis : A human perspective", Cell Stem Cell, 10, pp.120~136 (2012)[DOI]
68. Xiao Y, "Osteoclast precursors in murine bone marrow express CD27 and are impeded in osteoclast development by CD70 on activated immune cells", Proc Natl Acad Sci U S A, 110, pp.12385~12390 (2013)[DOI]
69. Ginhoux F, "The origin and development of nonlymphoid tissue CD103+ DCs", J Exp Med, 206, pp.3115~3130 (2009)[DOI]
70. Kingston D, "The concerted action of GM-CSF and Flt3-ligand on in vivo dendritic cell homeostasis", Blood, 114, pp.835~843 (2009)[DOI]
71. Sato K, "Osteoclasts, rheumatoid arthritis, and osteoimmunology", Curr Opin Rheumatol, 18, pp.419~426 (2006)[DOI]
72. Seeling M, "Inflammatory monocytes and Fcγ receptor IV on osteoclasts are critical for bone destruction during inflammatory arthritis in mice", Proc Natl Acad Sci U S A, 110, pp.10729~10734 (2013)[DOI]
73. Koizumi K, "Role of CX3CL1", fractalkine in osteoclast differentiation and bone resorption, 2009, pp.183, 7825~7831
74. Ibáñez L, "Inflammatory osteoclasts prime TNFα-producing CD4(+)T cells and express CX(3)CR1", J Bone Miner Res, 31, pp.1899~1908 (2016)[DOI]
75. Culemann S, "Locally renewing resident synovial macrophages provide a protective barrier for the joint", Nature, 572, pp.670~675 (2019)[DOI]
76. Hasegawa T, "Identification of a novel arthritis-associated osteoclast precursor macrophage regulated by FoxM1", Nat Immunol, 20, pp.1631~1643 (2019)[DOI]
77. Meirow Y, "Specific inflammatory osteoclast precursors induced during chronic inflammation give rise to highly active osteoclasts associated with inflammatory bone loss", Bone Res, 10, pp.36~ (2022)[DOI]
78. Madel MB, "Dissecting the phenotypic and functional heterogeneity of mouse inflammatory osteoclasts by the expression of Cx3cr1", Elife, 9, pp.e54493~ (2020)[DOI]
79. Strauss-Ayali D, "Monocyte subpopulations and their differentiation patterns during infection", J Leukoc Biol, 82, pp.244~252 (2007)[DOI]
80. Takeyama N, "Selective expansion of the CD14(+)", CD16(bright)subpopulation of circulating monocytes in patients with hemophagocytic syndrome, 2007, pp.86, 787~792
81. Todd I, "Elevated CD16 expression by monocytes from patients with tumor necrosis factor receptor-associated periodic syndrome", Arthritis Rheum, 56, pp.4182~4188 (2007)[DOI]
82. Ziegler-Heitbrock L, "The CD14+ CD16+ blood monocytes : their role in infection and inflammation", J Leukoc Biol, 81, pp.584~592 (2007)[DOI]
83. Frankenberger M, "Differential cytokine expression in human blood monocyte subpopulations : a polymerase chain reaction analysis", Blood, 87, pp.373~377 (1996)
84. Belge KU, "The proinflammatory CD14+CD16+DR++ monocytes are a major source of TNF", J Immunol, 168, pp.3536~3542 (2002)[DOI]
85. Feldmann M, "Development of anti-TNF therapy for rheumatoid arthritis", Nat Rev Immunol, 2, pp.364~371 (2002)[DOI]
86. Kawanaka N, "CD14+,CD16+ blood monocytes and joint inflammation in rheumatoid arthritis", Arthritis Rheum, 46, pp.2578~2586 (2002)[DOI]
87. Rossol M, "The CD14(bright)CD16+ monocyte subset is expanded in rheumatoid arthritis and promotes expansion of the Th17 cell population", Arthritis Rheum, 64, pp.671~677 (2012)[DOI]
88. Klimek E, "Blood monocyte subsets and selected cardiovascular risk markers in rheumatoid arthritis of short duration in relation to disease activity", Biomed Res Int, 2014, pp.736853~ (2014)[DOI]
89. Weldon AJ, "Surface APRIL is elevated on myeloid cells and is associated with disease activity in patients with rheumatoid arthritis", J Rheumatol, 42, pp.749~759 (2015)[DOI]
90. Tsukamoto M, "CD14(bright)CD16+ intermediate monocytes are induced by interleukin-10 and positively correlate with disease activity in rheumatoid arthritis", Arthritis Res Ther, 19, pp.28~ (2017)[DOI]
91. Yang J, "Monocyte and macrophage differentiation : circulation inflammatory monocyte as biomarker for inflammatory diseases", Biomark Res, 2, pp.1~ (2014)[DOI]
92. Yoon BR, "Functional phenotype of synovial monocytes modulating inflammatory T-cell responses in rheumatoid arthritis(RA)", PLoS One, 9, pp.e109775~ (2014)[DOI]
93. Winchester R, "Association of elevations of specific T cell and monocyte subpopulations in rheumatoid arthritis with subclinical coronary artery atherosclerosis", Arthritis Rheumatol, 68, pp.92~102 (2016)[DOI]
94. Veale DJ, "The pathogenesis of psoriatic arthritis", Lancet, 391, pp.2273~2284 (2018)[DOI]
95. Ortiz-Ruiz A, "Myc-related mitochondrial activity as a novel target for multiple myeloma", Cancers(Basel), 13, pp.1662~ (2021)[DOI]
96. Lomas OC, "The microenvironment in myeloma", Curr Opin Oncol, 32, pp.170~175 (2020)[DOI]
97. Sponaas AM, "The proportion of CD16(+)CD14(dim)monocytes increases with tumor cell load in bone marrow of patients with multiple myeloma", Immun Inflamm Dis, 3, pp.94~102 (2015)[DOI]
98. Bolzoni M, "IL21R expressing CD14(+)CD16(+)monocytes expand in multiple myeloma patients leading to increased osteoclasts", Haematologica, 102, pp.773~784 (2017)[DOI]
99. Petitprez V, "CD14+ CD16+ monocytes rather than CD14+ CD51", 61+ monocytes are a potential cytological marker of circulating osteoclast precursors in multiple myeloma. A preliminary study, 2015, pp.37, 29~35
100. Li X, "Maladaptive innate immune training of myelopoiesis links inflammatory comorbidities", Cell, 185, pp.1709~1727.e18 (2022)[DOI]
101. Locati M, "Diversity, mechanisms, and significance of macrophage plasticity", Annu Rev Pathol, 15, pp.123~147 (2020)[DOI]

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